No matter if you live in an apartment or house or just want to keep your home free of electromagnetic fields, there are a number options to limit your exposure. One of the easiest is to limit your electronic device use. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another way to shield your house against EMF radiations is to put up an RF shielding canopy. It is a type made of net which contains EMF shielding. It's used to stop EMFs from entering a room. Another option is to have your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
https://postheaven.net/hyenamimosa2/emf-block-paint-and-even-emf-shielding-35nj of studies have proven that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed be based on the downregulation in cancer-related stem cells. This could explain the long-term effects observed in some patients with advanced HCC. But, the reason for AM RF EMF's effect in cancer patients is not evident.
Aspects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumors were separated into three groups. The first group was not exposed RF EMF. Second group members were exposed RF EMF at frequencies similar to the frequency used by humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. https://telegra.ph/EMF-Block-Paint-plus-EMF-Shielding-04-14-35 of HCCMF on tumors was compared to that of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism of cancer-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have more broader implications for the treatment and diagnosis of various cancers.
The tumors in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumours in HCCMF HCCMF group were infected with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumors in the three groups showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, tumors that were treated using RCF didn't show reduction in size. The difference was significant. The tumors treated by RCF had necrosis that is common when tumors are that are exposed to RCF. There is a possibility that the necrosis was due to the lack of oxygen in the larger tumours.
In conclusion, the findings suggest the fact that AM EMF has anticancer effects in vitro and in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, https://cyclestraw4.werite.net/post/2023/04/14/EMF-Block-Paint-and-even-EMF-Shielding6 may have a long-lasting influence on the growth of HCC tumours in living tissue.
