If Additional reading reside in a house or apartment or just want to keep your home free of EMFs There are plenty options to limit your exposure. One of the easiest is to limit the use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another way to shield your home against EMF radiation is to put up an RF shielding canopy. This is a cloth of net that has EMF shielding. It's used to prevent EMFs from entering a room. Another alternative is to have your house equipped with a conductive enclosure. They are referred to as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This may account for the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients is not evident.
The effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. https://postheaven.net/hyenamimosa2/emf-blocking-plus-emf-shielding-2htc were divided into 3 groups. The first group was not exposed to RF EMF. Another group of participants was subjected RF EMF at the same frequency to the frequency used by humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, the tumours treated with RCF showed no evidence of shrinkage of the tumor.
The reason for tumour-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. http://beatmimosa6.xtgem.com/__xt_blog/__xtblog_entry/__xtblog_entry/33692335-emf-block-paint-and-even-emf-shielding?__xtblog_block_id=1#xt_blog upon HCC cells is caused by CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have more broader implications for diagnosis and treatment of a variety of cancers.
The tumours of those in the group that were unaffected RF EMF, and were fed a normal mouse diet. The tumors in HCCMF HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.
The tumors in the three groups also displayed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in size of the tumor after 8 weeks. However, tumors that were treated using RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF showed necrosis that is common in tumours that are exposed to RCF. There is a possibility that this necrosis was caused by a lack of oxygen in larger cancers.
In sum, the results show the fact that AM EMF has anti-cancer properties in vitro as well as in live. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in the vivo.